HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted Services Similar articles in this journal Alert me to new issues of the journal Download to citation manager Citing Articles Citing Articles via HighWire Citing Articles via Google Scholar Google Scholar Articles by Brown, E. A. Articles by Calvert, O. H. Search for Related Content PubMed Articles by Brown, E. A. Articles by Calvert, O. H. Agricola Articles by Brown, E. A. Articles by Calvert, O. H.

A Soybean Genotype Resistant Resistant to Phomopsis Seed Decay¹

Phomopsis seed decay (PSD) reduces soybean [Glyclne max (L.) Merr.| seed quality and seedling establishment. Experiments were conducted in Columbia, MO, and Isabela, PR, over a 3-yr period to identify a source(s) of resistance to PSD in soybean. All genotypes evaluated were field grown under conditions favorable for infection by Phomopsis spp. Percentage infection was determined by counting colonies of Phomopsis spp. growing from surface-sterilized seeds plated on acidified potato (Solanum spp.) dextrose agar. Plant introduction (PI) 417479 was free of Phomopsis spp. seed infection at Columbia in 1984 and Isabela in 1985, and had 3% infection at Columbia in 1985. A phenotypically indistinguishable genotype, PI 360841, had levels of infection that did not differ statistically from those observed for PI 417479 and may be a duplicate in the germplasm collection. In the same environments, the widely grown ‘Williams 82’ showed from 25 to 59% infection. The PI 417479 exhibited less infection than the reported P. sojae Lehman resistant genotypes PI 80837 and ‘Delmar’ in all but one test. Maturity dates of PI 417479 were near the average of all genotypes tested. The PI 417479 appears to have been exposed to average, or above average, disease pressure. Thus, the level of resistance to PSD measured cannot be attributed to maturation during periods of favorable environmental conditions. Also, antagonism of Phomopsis spp. seed infection by Cercospora kikuchii (T. Matsu. & Tomoyasu) Gardner did not appear to influence the conclusions relative to PI 417479. Detectable seed infection of PI 417479 by C. kikuchii was 2% or less in all experiments.

Key Words: Glycine max (L.) Merr. • Diaporthe spp. • Phomopsis spp. • Seed quality • PI 417479 • PI 360841

² Former graduate research assistant (currently soybean pathologist, J. Hartz Seed Co., Stuttgart, AR) and associate professor, Dep. of Agronomy, and professor, Dep. of Plant Pathology, Univ. of Missouri, Columbia, MO 65211, respectively.

Received for publication June 17, 1986.

This article has been cited by other articles:

				E. W. Jackson, P. Fenn, and P. Chen Inheritance of Resistance to Purple Seed Stain Caused by Cercospora kikuchii in PI 80837 Soybean Crop Sci., May 18, 2006; 46(4): 1462 - 1466. [Abstract] [Full Text] [PDF]

				E. W. Jackson, P. Fenn, and P. Chen Inheritance of Resistance to Phomopsis Seed Decay in Soybean PI 80837 and MO/PSD-0259 (PI 562694) Crop Sci., October 27, 2005; 45(6): 2400 - 2404. [Abstract] [Full Text] [PDF]